Chronic fluoxetine treatment induces structural plasticity and selective changes in glutamate receptor subunits in the rat cerebral cortex

Keyphrases

• Glutamate Receptors 100%
• Receptor Subunits 100%
• Rat Cerebral Cortex 100%
• Fluoxetine 100%
• Structural Plasticity 100%
• GluR1 100%
• GluR2 80%
• NR2A 80%
• Forebrain 40%
• Postsynaptic Density Protein 95 (PSD-95) 40%
• Structural Adaptation 40%
• Synaptic Membrane 40%
• Molecular Adaptation 40%
• Antidepressants 20%
• Spine 20%
• Immunohistochemistry 20%
• Synapse 20%
• Western Blot 20%
• Amino 20%
• Cortex 20%
• Propionate 20%
• Neuronal Network 20%
• Glutamatergic 20%
• Chronic Administration 20%
• Dendritic Spine Density 20%
• NR2B 20%
• Intracellular Membranes 20%
• Microsomal Fraction 20%
• Mushroom-like 20%
• Network Stabilization 20%
• Retrosplenial 20%
• Learned Helplessness 20%
• Stoichiometry 20%
• N-methyl-D-aspartate 20%
• Immunoprecipitates 20%
• Mood Improvement 20%
• Cerebral Cortex 20%

Neuroscience

• Glutamate Receptor 100%
• Receptor Subunit 100%
• Fluoxetine 100%
• Cerebral Cortex 100%
• Postsynaptic Density 66%
• Synaptic Membrane 66%
• Antidepressant 33%
• Immunohistochemistry 33%
• Immunoprecipitation 33%
• Synapse 33%
• Propionate 33%
• Protein Quaternary Structure 33%
• Dextro Aspartic Acid 33%
• Dendritic Spine 33%
• Propionic Acid 33%
• Intracellular Membrane 33%
• Vertebral Column 33%
• Receptor 33%
• Western Blotting 33%
• Learned Helplessness 33%